Studies aimed at both preventing the disease in at-risk pre-Type 1 subjects as well as intervening not only in new-onset cases, but also in established disease have been conducted over the past 30 years. Changing the course of the disease by preventing or slowing down beta cell destruction rather than managing hyperglycemia would significantly impact the burden. Glycemic targets both in children (and adolescents especially) as well as adults are met only by a minority of patients. Type 1a (autoimmune) diabetes remains a tremendous burden both to individuals and society.
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